[GUEST ACCESS MODE: Data is scrambled or limited to provide examples. Make requests using your API key to unlock full data. Check https://lunarcrush.ai/auth for authentication information.]  ☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆 [@DrJackKruse](/creator/twitter/DrJackKruse) on x 119.3K followers Created: 2025-07-20 12:32:02 UTC When tissues become a desert, they emit more light to cause collateral optical effects. We know that cancer is one effect of cell being more like Mars because CCO and the VDR receptor are non functional. We also know that cancers stimulate angiogensis. How? UPEs. Cancer angiogenesis, the formation of new blood vessels to support tumor growth, is primarily stimulated by hypoxia within the tumor due to it being a desert. CCO can no longer make enough water. This co morbid hypoxia triggers the release of massive arrays of UPEs that have the same spectra of pro-angiogenic factors, such as VEGF (vascular endothelial growth factor), which then signal nearby blood vessels to sprout and grow towards the tumor. The same process happens in mouth breathing. Mouth breathing also tells me something deep about the mammals in my office. I immediately order a brain MRI in a 3T magnet with my deuterium software program to look at the PONS, the floor of the fourth ventricle, and the RAS. Why? Where there is deuterium melanin needs renovation and often shows up. This is why I go look for melanin problems via MRI.Most mouth breathers are at risk for hemorrhagic CVA in the PONS, cerebellum, and brainstem. Many neurosurgeons (hint) following this should sit up in their chairs because the reason why humans bleed in those spots UNKNOWN is too centralized science, but I figured it out. No melanin = no H+, no oxygen, and no electrons from the charge separation of matrix water in those semiconductors in those X anatomic locations = massive UPE release to stimulate melanopsin damage and bleeding via hypoxia and VEGF. So as melanin degraded the endogenous light changes, the semiconductors there fail, light emission is changed inside of us and the non-visual photoreceptor melanopsin is disrupted. Did you know all cerebral blood vessels have melanopsin in them? Did you know melanopsin is the most common opsin in our heads? Now you know why brain bleeding happens and you might begin to understand really where aneurysms and AVM come from too!  XXXXXX engagements  **Related Topics** [mars](/topic/mars) [bitcoin](/topic/bitcoin) [coins layer 1](/topic/coins-layer-1) [coins bitcoin ecosystem](/topic/coins-bitcoin-ecosystem) [coins pow](/topic/coins-pow) [Post Link](https://x.com/DrJackKruse/status/1946910971244560731)
[GUEST ACCESS MODE: Data is scrambled or limited to provide examples. Make requests using your API key to unlock full data. Check https://lunarcrush.ai/auth for authentication information.]
☣️ Pleb Kruse = BTC foundationalist in exile 🟩🔆 @DrJackKruse on x 119.3K followers
Created: 2025-07-20 12:32:02 UTC
When tissues become a desert, they emit more light to cause collateral optical effects. We know that cancer is one effect of cell being more like Mars because CCO and the VDR receptor are non functional.
We also know that cancers stimulate angiogensis.
How?
UPEs.
Cancer angiogenesis, the formation of new blood vessels to support tumor growth, is primarily stimulated by hypoxia within the tumor due to it being a desert. CCO can no longer make enough water.
This co morbid hypoxia triggers the release of massive arrays of UPEs that have the same spectra of pro-angiogenic factors, such as VEGF (vascular endothelial growth factor), which then signal nearby blood vessels to sprout and grow towards the tumor.
The same process happens in mouth breathing.
Mouth breathing also tells me something deep about the mammals in my office. I immediately order a brain MRI in a 3T magnet with my deuterium software program to look at the PONS, the floor of the fourth ventricle, and the RAS. Why? Where there is deuterium melanin needs renovation and often shows up. This is why I go look for melanin problems via MRI.Most mouth breathers are at risk for hemorrhagic CVA in the PONS, cerebellum, and brainstem.
Many neurosurgeons (hint) following this should sit up in their chairs because the reason why humans bleed in those spots UNKNOWN is too centralized science, but I figured it out. No melanin = no H+, no oxygen, and no electrons from the charge separation of matrix water in those semiconductors in those X anatomic locations = massive UPE release to stimulate melanopsin damage and bleeding via hypoxia and VEGF.
So as melanin degraded the endogenous light changes, the semiconductors there fail, light emission is changed inside of us and the non-visual photoreceptor melanopsin is disrupted. Did you know all cerebral blood vessels have melanopsin in them?
Did you know melanopsin is the most common opsin in our heads? Now you know why brain bleeding happens and you might begin to understand really where aneurysms and AVM come from too!
XXXXXX engagements
Related Topics mars bitcoin coins layer 1 coins bitcoin ecosystem coins pow